
Pulmonary embolism: shock before hypotension
A maintained blood pressure may conceal deteriorating right ventricular function and organ perfusion in pulmonary embolism.
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A maintained blood pressure may conceal deteriorating right ventricular function and organ perfusion in pulmonary embolism.
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Classic ST elevation may be absent despite an occluded coronary artery requiring urgent reperfusion.
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Lifetime lipid exposure, inherited markers and inflammatory disease can change prevention decisions despite reassuring results.
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Atypical symptoms and absent risk factors do not reliably exclude myocardial infarction.
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Inotrope choice starts with perfusion, heart rate and afterload, not blood pressure alone.
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Wound location and ECG rhythm can sharpen thoracotomy decisions in traumatic cardiac arrest.
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Positive pressure changes preload and afterload, so its haemodynamic effect depends on the failing circulation.
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Troponin change identifies myocardial injury; mechanism, timing and imaging determine whether it is infarction.
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Bypass deliberately alters blood, temperature and coagulation, so abnormal numbers need physiological interpretation.
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A safer ED communication system separates urgent interruption from messages that can wait.
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Tamponade depends on impaired cardiac filling and haemodynamics, not simply the size of the effusion.
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After cardiac surgery, invasive data and procedure-specific responses can matter more than a generic arrest algorithm.
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