
When liver tests mislead in cirrhosis
Modest transaminases can mask worsening function, while INR alone cannot define haemostatic balance in cirrhosis.
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Modest transaminases can mask worsening function, while INR alone cannot define haemostatic balance in cirrhosis.
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Timing, trigger foods and absent skin or respiratory signs help distinguish FPIES from common mimics.
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Severe viral prodrome with rapid hypoxaemia needs rodent exposure, fluid restraint and early ECMO-capable escalation kept visible.
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An asymptomatic dilated ascending aorta needs whole-aorta imaging; sudden chest or back pain adds haemodynamic control and surgical discussion.
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Persistent ventricular fibrillation after three shocks needs a trained shock-vector plan before pauses and improvisation creep in.
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Abdominal distension with weight loss and high-protein ascites points beyond cirrhosis; raised ALT and childhood vaccines need structured checks.
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Persistent neonatal jaundice needs fractionated bilirubin before a breast milk jaundice explanation; chest pain ECGs and respiratory failure add urgent checks.
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Chronic red legs need heat, laterality and elevation checks before antibiotics; aspiration-related nodules and haemorrhage control add higher-acuity lessons.
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Collapse that looks settled still needs ECG-led syncope risk assessment before haemorrhagic ascites and MOGAD steroid decisions follow.
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Dyspepsia, sudden desaturation, and the communication habits that change clinical decisions.
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